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ACCESSIONPLANT NAMETAXONOMYORIGINGENEBANKIMAGEAVAILABILITYRECEIVEDSOURCE TYPESOURCE DATECOLLECTION SITECOORDINATESELEVATIONHABITATIMPROVEMENT LEVELNARRATIVE
0PI 657941SCM(3)-4-3Gossypium hirsutum L. Mississippi, United StatesCOTNot Available2009DEVELOPED2009Genetic materialAs the imazamox rates increased, lint yields of the tolerant mutants decreased only gradually whereas yields of the susceptible non-mutant cultivars decreased drastically. In 2005, application of 88 g a.i. ha-1 imazamox reduced lint yield of tolerant mutants by 2 % compared to 41 % for the non-mutated susceptible cultivars. In 2006, this rate reduced lint yield in the tolerant mutants by 3 % whereas 25 % lint reduction was observed in the susceptible cultivars. Highly significant differences between the different cultivars for fiber length and strength were observed in both 2005 and 2006. Fiber length showed significant difference between imazamox doses only in 2005 and fiber strength difference between the different doses were significant only in 2006. Dose by cultivar interactions were not significant for any of the quality parameters in both 2005 and 2006. F2 populations resulting from susceptible x tolerant crosses gave a good fit to a 1:2:1 T/I/S ratio indicating segregation of a single gene for resistance to imazamox. BC1F1 populations gave a good fit to a 1:1 I/S ratio, confirming the single locus hypothesis. Allelism tests were conducted by crossing the four mutants among themselves. No intermediate or susceptible progeny were observed in the F2 of all the crosses. The tolerant genes in the four mutants are therefore either alleles at the same locus or very tightly linked. DNA fingerprinting analysis indicated genetic differences between the mutants and their original parents.1812160PI 657941
1PI 657942SCM(3)-7-3Gossypium hirsutum L. Mississippi, United StatesCOTNot Available2009DEVELOPED2009Genetic materialAs the imazamox rates increased, lint yields of the tolerant mutants decreased only gradually whereas yields of the susceptible non-mutant cultivars decreased drastically. In 2005, application of 88 g a.i. ha-1 imazamox reduced lint yield of tolerant mutants by 2 % compared to 41 % for the non-mutated susceptible cultivars. In 2006, this rate reduced lint yield in the tolerant mutants by 3 % whereas 25 % lint reduction was observed in the susceptible cultivars. Highly significant differences between the different cultivars for fiber length and strength were observed in both 2005 and 2006. Fiber length showed significant difference between imazamox doses only in 2005 and fiber strength difference between the different doses were significant only in 2006. Dose by cultivar interactions were not significant for any of the quality parameters in both 2005 and 2006. F2 populations resulting from susceptible x tolerant crosses gave a good fit to a 1:2:1 T/I/S ratio indicating segregation of a single gene for resistance to imazamox. BC1F1 populations gave a good fit to a 1:1 I/S ratio, confirming the single locus hypothesis. Allelism tests were conducted by crossing the four mutants among themselves. No intermediate or susceptible progeny were observed in the F2 of all the crosses. The tolerant genes in the four mutants are therefore either alleles at the same locus or very tightly linked. DNA fingerprinting analysis indicated genetic differences between the mutants and their original parents.1812161PI 657942
2PI 657943RM(3)-8-1Gossypium hirsutum L. Mississippi, United StatesCOTNot Available2009DEVELOPED2009Genetic materialAs the imazamox rates increased, lint yields of the tolerant mutants decreased only gradually whereas yields of the susceptible non-mutant cultivars decreased drastically. In 2005, application of 88 g a.i. ha-1 imazamox reduced lint yield of tolerant mutants by 2 % compared to 41 % for the non-mutated susceptible cultivars. In 2006, this rate reduced lint yield in the tolerant mutants by 3 % whereas 25 % lint reduction was observed in the susceptible cultivars. Highly significant differences between the different cultivars for fiber length and strength were observed in both 2005 and 2006. Fiber length showed significant difference between imazamox doses only in 2005 and fiber strength difference between the different doses were significant only in 2006. Dose by cultivar interactions were not significant for any of the quality parameters in both 2005 and 2006. F2 populations resulting from susceptible x tolerant crosses gave a good fit to a 1:2:1 T/I/S ratio indicating segregation of a single gene for resistance to imazamox. BC1F1 populations gave a good fit to a 1:1 I/S ratio, confirming the single locus hypothesis. Allelism tests were conducted by crossing the four mutants among themselves. No intermediate or susceptible progeny were observed in the F2 of all the crosses. The tolerant genes in the four mutants are therefore either alleles at the same locus or very tightly linked. DNA fingerprinting analysis indicated genetic differences between the mutants and their original parents.1812162PI 657943
3PI 657944EM(4)-3-1Gossypium hirsutum L. Mississippi, United StatesCOTNot Available2009DEVELOPED2009Genetic materialAs the imazamox rates increased, lint yields of the tolerant mutants decreased only gradually whereas yields of the susceptible non-mutant cultivars decreased drastically. In 2005, application of 88 g a.i. ha-1 imazamox reduced lint yield of tolerant mutants by 2 % compared to 41 % for the non-mutated susceptible cultivars. In 2006, this rate reduced lint yield in the tolerant mutants by 3 % whereas 25 % lint reduction was observed in the susceptible cultivars. Highly significant differences between the different cultivars for fiber length and strength were observed in both 2005 and 2006. Fiber length showed significant difference between imazamox doses only in 2005 and fiber strength difference between the different doses were significant only in 2006. Dose by cultivar interactions were not significant for any of the quality parameters in both 2005 and 2006. F2 populations resulting from susceptible x tolerant crosses gave a good fit to a 1:2:1 T/I/S ratio indicating segregation of a single gene for resistance to imazamox. BC1F1 populations gave a good fit to a 1:1 I/S ratio, confirming the single locus hypothesis. Allelism tests were conducted by crossing the four mutants among themselves. No intermediate or susceptible progeny were observed in the F2 of all the crosses. The tolerant genes in the four mutants are therefore either alleles at the same locus or very tightly linked. DNA fingerprinting analysis indicated genetic differences between the mutants and their original parents.1812163PI 657944